A regulatory instead of an IL-17 T response predominates in Helicobacter pylori-associated gastritis in children.

نویسندگان

  • Fabrício Freire de Melo
  • Andreia Maria Camargos Rocha
  • Gifone Aguiar Rocha
  • Silvia Helena Sousa Pietra Pedroso
  • Sérgio de Assis Batista
  • Lúcia Porto Fonseca de Castro
  • Simone Diniz Carvalho
  • Paulo Fernando Souto Bittencourt
  • Celso Afonso de Oliveira
  • Rodrigo Corrêa-Oliveira
  • Dulciene Maria Magalhães Queiroz
چکیده

Th17 cells seem to have an important role in the efficacy of vaccines against Helicobacter pylori. Because children are a target group for human vaccination and Th17/T(reg) cells have intrinsically linked and antagonic commitments, we compared the gastric levels of Th17- and T(reg)-associated cytokines of children and adults. IL-6, IL-10 and TGF-β1 levels and Foxp3(+) cell numbers were higher, but IL-1β, IL-17A and IL-23 were lower in infected children than in infected adults. In conclusion T(reg) instead of Th17 cell response to H. pylori-infection predominates in children.

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

ENDOSCOPIC NODULAR GASTRITIS: AN INDICATOR OF HELICOBACTER PYLORI INFECTION IN CHILDREN

Objective: To investigate the importance of endoscopic nodular gastritis associated With Helicobacter pylori infection in children. Methods: In this prospective study, 220 consecutive patients (age range 2 to 15 years, mean age 9. 02 ±3 .3 ), 118 of them males (53. 6%) underwent upper endoscopy during evaluation of chronic abdominal pain. The appearance of nodular gastritis was observed du...

متن کامل

Association between Helicobacter pylori cagA, babA2 Virulence Factors and Gastric Mucosal Interleukin-33 mRNA Expression and Clinical Outcomes in Dyspeptic Patients

Helicobacter pylori (H. pylori) infection has been reported in more than half of the world human population. It is associated with gastric inflammation and noticeable infiltration of the immune cells to the stomach mucosa by several cytokines secretion. IL-1&beta, IL-18 have been shown to contribute to H. pylori induced gastritis, but the details of inflammation and association of virulence fac...

متن کامل

Serum levels of iron parameters and IL-17 in children with Helicobacter pylori infection compared to healthy group

Helicobacter pylori is related to iron deficiency anemia (IDA) and inflammatory responses causing gastric and duodenal ulcer and carcinoma. Moreover, it leads to deficiency of factors associated with iron adsorption and transfer. In the present study, we recruited 100 children (50 infected with H. pylori and 50 controls) aged 3-14 years old (40% male and 60% female) to evaluate the effect of H....

متن کامل

IL-23 Contributes to Control of Chronic Helicobacter Pylori Infection and the Development of T Helper Responses in a Mouse Model1

The immune response to Helicobacter pylori involves a mixed T helper-1, T helper-2, and T helper-17 response. It has been suggested that T helper cells contribute to the gastric inflammatory response during infection, and that T helper 1 (Th1) and T helper 17 (Th17) subsets may be required for control of H. pylori colonization in the stomach. The relative contributions of these subsets to gastr...

متن کامل

Immune Responses Mediated by Th17 Cells in Helicobacter pylori Infection

T helper 17 (Th17) cells are one of the CD4+ T-cell subsets which induce a variety of diseases by secreting IL-17 and other inflammatory factors. After Helicobacter pylori (Hp) infection, cytotoxin-associated gene A and urease subunit B regulate the number of Th17 cells via induction of cell differentiation by infected macrophages, activation of MyD88 and other pathways, and by driving chemokin...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:
  • Microbes and infection

دوره 14 4  شماره 

صفحات  -

تاریخ انتشار 2012